Author(s) |
(Date) |
Paper Title |
Citation |
Model Type |
Applications |
Full Text |
| Toledo F, et al. Mouse over for full list Toledo F, Liu CW, Lee CJ, Wahl GM |
(2006) | RMCE-ASAP: a gene targeting method for ES and somatic cells to accelerate phenotype analyses. | Nucleic Acids Res. 34(13):e92 | |||
| Pritchard JB, et al. Mouse over for full list Pritchard JB, French JE, Davis BJ, Haseman JK |
(2003) | The role of transgenic mouse models in carcinogen identification. | Environ. Health Perspect. 111(4):444-54 | |||
| Johansson FK, et al. Mouse over for full list Johansson FK, Brodd J, Eklöf C, Ferletta M, Hesselager G, Tiger CF, Uhrbom L, Westermark B |
(2004) | Identification of candidate cancer-causing genes in mouse brain tumors by retroviral tagging. | Proc. Natl. Acad. Sci. U.S.A. 101(31):11334-7 | Oncology | ||
| Horn TL, et al. Mouse over for full list Horn TL, Cwik MJ, Morrissey RL, Kapetanovic I, Crowell JA, Booth TD, McCormick DL |
(2007) | Oncogenicity evaluation of resveratrol in p53(+/-) (p53 knockout) mice. | Food Chem. Toxicol. 45(1):55-63 | Oncology | ||
| Ferguson DO, et al. Mouse over for full list Ferguson DO, Sekiguchi JM, Chang S, Frank KM, Gao Y, DePinho RA, Alt FW |
(2000) | The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations. | Proc. Natl. Acad. Sci. U.S.A. 97(12):6630-3 | |||
| Donehower LA, et al. Mouse over for full list Donehower LA, Harvey M, Slagle BL, McArthur MJ, Montgomery CA, Butel JS, Bradley A |
(1992) | Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours. | Nature 356(6366):215-21 | |||
| Bielas JH, et al. Mouse over for full list Bielas JH, Heddle JA |
(2003) | Elevated mutagenesis and decreased DNA repair at a transgene are associated with proliferation but not apoptosis in p53-deficient cells. | Proc. Natl. Acad. Sci. U.S.A. 100(22):12853-8 |
